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The MedStar Family Choice P&T Committee met in October 2007, reviewed the entire formulary and made changes to the 2008 formulary. Medications that were removed are: Cretor rosuvastatin calcium ; Azmacort triamcinolone acetonide ; Members who are currently prescribed these medications will be "grandfathered" and will be able to continue to fill these medications as usual. Starting January 1, 2008, first time prescriptions for patients not previously prescribed these medications will be considered requests for non-formulary medications!

To be sure, one week's data on prescription patterns is too little to draw broad conclusions. The ImpactRx numbers reflect only prescriptions written for newly diagnosed patients, or for patients who switch to different drugs -- generally about 10% of the total number of prescriptions issued, said Fred Nelson, head of business development, marketing and public relations at the company. But the initial response does signal the challenge AstraZeneca faces to win converts to its drug. The sharp decline came despite evidence from ImpactRx data that AstraZeneca's sales force was making more calls on doctors than any of its competitors. Beginning in late February, reflecting the sales calls, new prescriptions of C4estor began to rise and overtook Lipitor by the beginning of March. Then Ctestor lost some ground after the Public Citizen news before falling off sharply in the wake of the study results. Needs Data "Crestor is a very strong drug, but it doesn't really have much hard data to go for it, " said George Dangas, a cardiologist at Lenox Hill Heart and Vascular Institute, New York. He referred to the lack of studies showing that Creztor prevents heart attacks and death. He was also impressed that few patients in the Prove-It trial, which tested high-dose Lipitor, had significant side effects.
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However, steroids can cause numerous side effects, including bleeding from the stomach, loss of calcium from bones, cataracts in the eye, and a diabetes-like state and hytrin. Pain, watery diarrhea, and jaundice are present leading to misdiagnosis of viral gastroenteritis or hepatitis. Clinical manifestations are associated with the adherence of parasitized red blood cells in the microvasculature of the gastrointestinal tract. Anemia. Destruction of red blood cells upon merozoite release, and inhibition of hematopoesis by tissue necrosis factor- cause the severe anemia often seen in P. falciparum infections. Also, P. falciparum parasites can infect red blood cells of all ages, which theoretically allows infection of all circulating red blood cells. Whereas, P. vivax and P. ovale require young red blood cells reticulocytes ; and P. malariae requires mature blood cells for infection. Severe anemia is defined as a hematocrit of less than 21%, and clinical manifestations may include dark brown or red urine hemoglobinuria ; , decrease in urine production, and jaundice. Renal failure, as previously discussed, may be a complication. Another cause of hemolysis and hemoglobinuria in patients with malaria is destruction of G-6-PD deficient red blood cells by oxidant antimalarial drugs such as primaquine. Malaria due to P. vivax or P. ovale ; Infection Infections due to P. vivax and P. ovale are virtually the same. Both are less severe than falciparum malaria, and parasite blood levels are lower. Parasitized red blood cells do not develop knobs, therefore no microvascular obstruction with resultant brain, kidney, lung, or other organ complications occur in malaria due to P. vivax or P. ovale. P. vivax and P. ovale form a dormant stage in liver cells called hypnozoites. These parasites activate and cause delayed infections or a relapse Table 3-3 ; . A relapse usually occurs within 6 months of an acute attack. Some hypnozoites remain dormant much longer, and are virtually undetectable. If there is any suspicion that P. vivax or P. ovale is endemic in the area of exposure, presumptive treatment must be given to prevent illness. Currently, the only available treatment is primaquine; dosages are listed in Chapters 4, 5, and Appendix 4. As previously stated, fever is virtually always present, and fever plus any other symptom might be malaria. P. vivax or P. ovale fevers may be erratic or continuous in the initial phase of illness. After 3 to.

Health care workers are at high risk for exposure to natural rubber latex NRL ; and commercial cleaning solutions; risk assessment and appropriate control measures should be implemented to minimize exposure to allergens and irritants and to reduce the risk of workers for developing occupational asthma. For sensitized individuals, special attention should be given to their work and home environments, because timely control of exposure is important to minimize further damage and long-term adverse effects and innopran.

Bma - gms2 update, 8 march 2004 1 - jump to homepage 3 - jump to site map 4 - jump to search 9 - jump to contact us 0 - jump to accessibility statement s - jump to main content n - jump to navigation menu site map contact us about us how to join feedback advanced search browse pages by term back to home back to archive back to archive - general practitioners back to 2004 archived documents you are here: home archive archive - general practitioners 2004 archived documents gms2 update 8 march 2004 enhanced services financial monitoring of enhanced services focus on. Article outline abstract women's health oxytrol valtrex femring men's health uroxatral levitra immunologic flumist asthma update xolair dermatology raptiva cubicin cardiovascular: crestor coreg devices lower cost defibrilator combination diabetes device new blood test for chd risk new mammography device references citing articles figures tables figure and atacand. H E SYDDALL1 A AIHIE SAYER1, 2, E M DENNISON1, H J MARTIN1, C COOPER1 & THE HERTFORDSHIRE COHORT STUDY GROUP 1 ; MRC Epidemiology Resource Centre, University of Southampton and 2 ; University Geriatric Medicine, University of Southampton Introduction The Hertfordshire Cohort Study HCS ; comprises 3, 000 men and women born in Hertfordshire 1931-39, for whom birth weight and infant development was recorded by midwives; and whose adult diet and lifestyle, and prevalence of cardiovascular disease, osteoporosis, sarcopenia and other chronic disorders has been assessed recently. The representativeness of this cohort, and response bias effects, are unknown. Methods We compared the socio-economic, anthropometric, medical and functional characteristics of participants in HCS and the nationally representative Health Survey for England HSE ; . We assessed response bias between the HCS home interview and clinic. Results HCS and HSE participants were broadly comparable although HCS participants were less likely to be in the extremes of the socio-economic distribution, were taller, had better self-reported general health excellent very good, HCS vs HSE: men 54.3% vs 36.2%, women 43.8% vs 33.0% ; , and HCS women were less likely to be current smokers 11.5% vs 22.5% ; or heavy drinkers 13.2% vs 23.8% 8 units weekly ; . HCS clinic participants smoked less ever smokers: 67.7% men, 39.6% women ; than those only completing the home interview 80.7% men, p 0.06; 53.8% women, p 0.003 ; , and had better self-reported health excellent to good: 89.9% vs 64.6% men, p 0.001; 83.7% vs 71.8% women, p 0.03 ; . Conclusion HCS results can be reasonably generalised to the wider population of older people in England. In common with many epidemiological studies, a 'healthy' responder bias was evident in HCS. Segments of colon, ments that require thus enhancing and gist's image analysis and lopid.

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Is that without chemotherapy, my liver function is certainly not improving. With a prevalence of 5% to 15%, atopic dermatitis is the most common dermatitis seen in childhood. Most patients will develop symptoms of this pruritic eruption before the age of two with facial, extensor, or generalized involvement in infants. The distribution evolves into flexural and palmoplantar involvement in adolescents.The exact etiology of this condition is unknown although there is currently no evidence to suggest food or other allergies as the cause in most children. Many patients will have associated asthma and seasonal allergies with a family history of these same conditions. Other cutaneous features associated with atopic dermatitis include xerosis, ichthyosis vulgaris, keratosis pilaris, pityriasis alba, and hyperlinear palmar creases. Treatment for atopic dermatitis lies in decreasing inflammation, treating coexisting infections, restoring the barrier function of the skin, and controlling any exacerbating factors. Moisturization of the skin will help to restore the skin barrier function and increase the rate of healing. Creams or ointments, such as petroleum jelly, are very effective in hydrating the skin and should be used after a bath and liberally throughout the day. Atopic patients often benefit from the use of mild soaps or soap substitutes. Use of a humidifier in the home and wet wraps during the dry winter months will improve skin hydration. Topical corticosteroids remain one of the mainstays of treatment. In children, daily use of mid-potency steroids for the body and low-potency steroids for the face when needed will help to decrease inflammation in the skin. In areas of lichenification, mid- to highpotency steroids for two weeks may be necessary. Although messy to use, topical tar preparations are also effective and lotensin. Freshly drawn human blood was incubated with 2.5% gelatin in saline 1: ratio ; for 30 min at 37C. The supernatant was layered on Histopaque 1077 from Sigma ; and centrifuged at 1500 rpm for 30 min at room temperature. The cells were then collected from the top layer of Histopaque, diluted with Dulbecco's PBS, and centrifuged at 2000 rpm for 10 min. To get rid of mixed reticulocytes, pellet was suspended in 0.2% NaCl for 1 min, immediately diluted with equal volume of 1.6% NaCl, and centrifuged at 1000 rpm. To remove macrophages by adherence, the pellet was suspended in RPMI 1640 medium supplemented with 10% FBS and cultured for 2 h at 37C, CO2 incubator in a petri dish. Then the lymphocytes were harvested from the medium by centrifugation at 1000 rpm.
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Homozygous Familial Hypercholesterolemia In an open-label, forced-titration study, homozygous FH patients n 40, 8-63 years ; were evaluated for their response to CRESTOR 20 to 40 mg titrated at a 6-week interval. In the overall population, the mean LDL-C reduction from baseline was 22%. About one-third of the patients benefited from increasing their dose from 20 mg to 40 mg with further LDL lowering of greater than 6%. In the 27 patients with at least a 15% reduction in LDL-C, the mean LDL-C reduction was 30% median 28% reduction ; . Among 13 patients with an LDL-C reduction of 15%, 3 had no change or an increase in LDL-C. Reductions in LDL-C of 15% or greater were observed in 3 of patients with known receptor negative status. INDICATIONS AND USAGE CRESTOR is indicated: 1. as an adjunct to diet to reduce elevated total-C, LDL-C, ApoB, nonHDL-C, and TG levels and to increase HDL-C in patients with primary hypercholesterolemia heterozygous familial and nonfamilial ; and mixed dyslipidemia Fredrickson Type IIa and IIb 2. as an adjunct to diet for the treatment of patients with elevated serum TG levels Fredrickson Type IV 3. to reduce LDL-C, total-C, and ApoB in patients with homozygous familial hypercholesterolemia as an adjunct to other lipid-lowering treatments e.g., LDL apheresis ; or if such treatments are unavailable. According to NCEP-ATPIII guidelines, therapy with lipid-altering agents should be a component of multiple-risk-factor intervention in individuals at increased risk for coronary heart disease due to hypercholesterolemia. The two major modalities of LDL-lowering therapy are therapeutic lifestyle changes TLC ; and drug therapy. The TLC Diet stresses reductions in saturated fat and cholesterol intake. Table 5 defines LDL-C goals and cutpoints for initiation of TLC and for drug consideration. Bodies. In preparation for the adoption of new international accounting standards in 2005, AstraZeneca was the first FTSE 100 company to make available to shareholders financial information for 2003 and the first half of 2004 prepared in accordance with the new standards. AstraZeneca's share price performance, and that of other major pharmaceutical companies, were disappointing in 2004 with the AstraZeneca share price in particular affected by the FDA's nonapproval of Exanta, the challenges facing Crest0r and the recent clinical trial results for Iressa. The composition of the Board is also undergoing some change. On my retirement at the end of the year, the Board confirmed the appointment of Louis Schweitzer as my successor as NonExecutive Chairman of AstraZeneca with effect from 1 January 2005, following his appointment to the Board in March 2004. Louis Schweitzer is a distinguished industrialist with wide international experience and I congratulate him most warmly on his appointment. Karl von der Heyden, the Chairman of the Audit Committee, retired at the 2004 AGM after more than five years as a Non-Executive Director. I thank him for his contribution to the Company and, in particular, the role he played in the development of the work of the Audit Committee. John Buchanan succeeded Karl as Chairman of the Audit Committee. Most recently, the Board announced the appointment of Dr John Patterson, with effect from 1 January 2005, to the Board as Executive Director responsible for Development, emphasising the importance we place on this activity and micardis and Crestor online.

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Hypertriglyceridemia fredrickson type iib & iv ; in a double blind, placebo-controlled dose-response study in patients with baseline tg levels from 273 to 817 mg dl, crestor given as a single daily dose 5 to 40 mg ; over 6 weeks significantly reduced serum tg levels table 4 and zocor.

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Group of study participants capable of long-term protocol compliance. Following the run-in period, participants will be randomly assigned to either oral rosuvastatin 20 mg d; supplied as CRESTOR by AstraZeneca [Wilmington, Del] ; or placebo for a period of 3 to years, the estimated time needed to accrue the 520 cardiovascular end points on which the study is powered. The dose of rosuvastatin selected should result in 50% reductions in LDL cholesterol57 as well as a substantial reduction in hsCRP. All study participants will visit the clinic sites for evaluation at 3 and 6 months after randomization and thereafter at 6-month intervals for the duration of follow-up. At these visits, staff will dispense study medication; assess compliance with pill taking, the use of concomitant medications, and the development of major illnesses, study end points, or adverse effects; and collect fasting blood and urine samples to evaluate changes in lipid and inflammatory parameters and to monitor safety. Study medication will be discontinued among subjects who develop myopathy CK 10 times ULN and muscle aches or weakness ; or a persistent elevation in ALT 3 times ULN on 2 consecutive tests ; . Subjects whose blinded LDL-C levels rise to 130 mg dL during the study will be counseled to adopt lifestyle changes recommended by the NCEP. If, after 3 months, LDL-C levels remain elevated and the calculated Framingham Risk Score exceeds 10% despite lifestyle changes, investigators will be encouraged to consider lipid-lowering therapy with bile-acid sequestrants or cholesterol-absorption inhibitors for those subjects. However, if the responsible study physician believes statin therapy is indicated, the study medication will be discontinued and open-label statin therapy will be initiated. All subjects in whom study medication is discontinued will be followed for the duration of the trial and included in data analyses.
10 mg, 20 mg, 40 mg Film-Coated Tablets COMPOSITION : Each tablet contains 10 mg, 20 mg, or 40 mg of rosuvastatin as rosuvastatin calcium. PHARMACEUTICAL FORM : Film-coated tablet. Round, pink coloured 10 mg and 20 mg oval, pink coloured 40 mg ; . THERAPEUTIC INDICATIONS : Primary hypercholesterolaemia type IIa including heterozygous familial hypercholesterolaemia ; or mixed dyslipidaemia type IIb ; as an adjunct to diet when response to diet and other non-pharmacological treatments e.g. exercise, weight reduction ; is inadequate. Homozygous familial hypercholesterolaemia as an adjunct to diet and other lipid lowering treatments e.g. LDL apheresis ; or if such treatments are not appropriate. POSOLOGY AND METHOD OF ADMINISTRATION : Before treatment initiation the patient should be placed on a standard cholesterol-lowering diet that should continue during treatment. The dose should be individualised according to the goal of therapy and patient response, using current consensus guidelines. The recommended start dose is 10 mg orally once daily and the majority of patients are controlled at this dose. A dose adjustment to 20 mg can be made after 4 weeks, if necessary see Pharmacodynamic properties ; . A doubling of the dose to 40 mg should only be considered in patients with severe hypercholesterolaemia at high cardiovascular risk in particular those with familial hypercholesterolaemia ; , who do not achieve their treatment goal on 20 mg, and in whom routine follow-up will be performed see Special warnings and precautions ; . Crestor may be given at any time of day, with or without food. Paediatric use Safety and efficacy have not been established in children. Paediatric experience is limited to a small number of children aged 8 years or above ; with homozygous familial hypercholesterolaemia. Therefore, Crestor is not recommended for paediatric use at this time. Use in the elderly No dose adjustment is necessary. Dosage in patients with renal insufficiency. No dose adjustment is necessary in patients with mild to moderate renal impairment. The use of Crestor in patients with severe renal impairment is contraindicated see Contraindications and Pharmacokinetic properties ; . Dosage in patients with hepatic impairment. There was no increase in systemic exposure to rosuvastatin in subjects with Child-Pugh scores of 7 or below. However, increased systemic exposure has been observed in subjects with Child-Pugh scores of 8 and 9 see Pharmacokinetic properties ; . In these patients an assessment of renal function should be considered see Special warnings and special precautions for use ; . There is no experience in subjects with Child-Pugh scores above 9. Crestor is contraindicated in patients with active liver disease see Contraindications.

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Complementary therapies for HIV have been defined as the `scientific and non-scientific interventions that diverge from the accepted traditional treatment of HIV infection.'1 Recent reports indicate that complementary and alternative medicine CAM ; use among North American HIV patients may be as high as 70%, 24 and that HIV patients do not communicate CAM usage to their physicians.5 HIV patients in a recent qualitative study believed that complementary therapies would be synergistic with antiretrovirals or reduce HIV-related symptoms.6 Physicians treating HIV patients need to be aware of their patients' use of CAM and the potential for CAM therapies to influence the response to conventional treatments. Previous studies suggesting that natural health products NHPs ; could interact with HIV medications and lead to drug failure and resistance7 have received widespread public health attention.8, 9. We thank Dr. Robert A. Rieger for electron spray ionization MS data collection and Dr. Melanie Nilsson for performing initial HPLC analysis of the INH-NAD adduct. This work was supported by National Institutes of Health Grant AI44639 to P.J.T. ; . P.J.T. is an Alfred P. Sloan Fellow and buy diovan.
Answer: since, you experienced pain in your hands and calves when you were on lipitor atorvastatin ; , neither zocor simvastatin ; nor crestor rosuvastatin ; are likely to be good alternatives. If you need to, buy more life insurance now since it takes time to get a policy. If nothing happens, you can always cancel it later. You may wish to consider buying a life insurance policy for your spouse and children. It would be prudent to select only the bluest of blue chip insurers, as the economic impact of a major pandemic will not be predictable. Also, if a pandemic happens and a lot of folks die, the cost of life insurance in the future will be higher. Tidbits From The Chemical Feast: You might want to keep this guide to 7 different cardiovascular medications: Statins: inhibit the production of cholesterol in the liver, reduce inflammation, and may protect the heart through other mechanisms. Examples: atorvastin lipitor ; , simvastatin zocor ; , pravastatin pravachol ; , rosuvastatin crestor ; . Side effects: nausea or upset stomach, elevation in liver enzymes uncommon ; . Muscle soreness may precede a condition called rhabdomyolysis which may inflict muscle damage which can lead to kidney failure but get a creatinine test via your doctor before you jump to conclusions. Two tupolev aircraft from the early 1930s, the giant, eig.

And, by coerced i mean when my husband applied for life insurance and the company contacted his doctor, the doctor told them he wouldn't take the medicines prescribed to him so the insurance oompany turned him down.
Good morning, I'm Dr. An Pham, medical assigned liaison in cardiovascular with Schering Plough. I would like to thank the P&T committee for the opportunity to provide public comment on the clinical benefit of Vytorin for the treatment of elevated LDL cholesterol for many high risk patients in Washington. As you know, LDL cholesterol comes from three sources, their food, the liver, and the [inaudible]. So the question is how it's important and clinically relevant to treat these three sources of cholesterol, especially in high risk patients, including patients with diabetes and CHD [inaudible]. As an analogy, if your office is in a high risk, tough neighborhood and your office has three doors, is it enough to lock just one door? Or all three doors together at the end of the day? Vytorin, a powerful lipid lowering therapy is available in a once daily tablet. Vytorin containing ezetimibe and Simvastatin is the first and only first line lipid lowering agent approved to simultaneously treat the primary sources of hypercholesterolemia. First by inhibiting the production of cholesterol in the liver through [inaudible] statin and secondly by uniquely blocking the absorption of cholesterol from [inaudible] and dietary in the small intestine. The complimenting mechanism of Vytorin reduced LDL-c by 52% of its usual 10, 20 starting dose. Furthermore, Vytorin is indicated to increased HDL and lower triglyceride. In the three large head-to-head studies as presented by Dr. McDonagh, Vytorin demonstrates the pure efficacy to both Lipitor and Crestor in achieving mean LDL-c cholesterol across the entire dosing range and superior in the percentage of patients attaining the NCEP lipid goals of less than 70 for LDL cholesterol for high risk patients, including the type 2 diabetic or CHD equivalent. More importantly, several other large outcome studies are currently underway to assess the additional benefit of Vytorin on cardiovascular morbidity and mortality above and beyond that demonstrated for Simvastatin. Most importantly, Vytorin is generally well tolerated and has not demonstrated an adverse event profile significantly different in quantity or scope than statin alone. Please refer to the full prescribing information for additional information. In summary, Vytorin in a single tablet offers patients superior lipid lowering efficacy and helps many high risk patients to achieve the new NCEP guideline treatment goal of less than 70 for LDL cholesterol. Thank you very much for the consideration and I would strongly ask the P&T committee to place Vytorin on the preferred status of Washington State Medicaid for the benefit of patient access.
Dear Health Care Professional, AstraZeneca Canada Inc., in consultation with Health Canada, would like to inform you of important safety information for CRESTOR rosuvastatin ; . CRESTOR is a lipid-lowering agent of the statin class, which has been available in Canada since February 2003. In June 2004, AstraZeneca Canada Inc., in consultation with Health Canada, issued a letter to Canadian health care professionals advising of an association between use of CRESTOR rosuvastatin ; and rhabdomyolysis. A second advisory was posted to the Health Canada website in November 2004 identifying an increased reporting rate of rhabdomyolysis associated with use of the CRESTOR 40 mg daily dose. Health Canada has conducted further review of safety information relating to use of CRESTOR, including Canadian adverse event reports, and has requested changes to the Canadian Product Monograph for CRESTOR rosuvastatin ; to permit the safe use of this medication.
Payment will be denied for patients taking 1 dosage units per day for the following agents and respective strengths: o atorvastatin lipitor ; 10mg, 20mg, 40mg o fluvastatin lescol ; 20mg, 40mg o lovastatin mevacor ; 10mg, 20mg o pravastatin pravachol ; 10mg, 20mg, 40mg o rosuvastatin crestor ; 5mg, 10mg, 20mg o simvastatin zocor ; 5mg, 10mg, 20mg.
Collaboration with Bristol-Myers Squibb Company to develop and commercialise two investigational compounds being studied for the treatment of Type 2 diabetes, saxagliptin and dapagliflozin. Collaboration with POZEN Inc. to develop a fixed dose combination of naproxen and esomeprazole for chronic pain PN400 ; , utilising POZEN's proprietary technology. Agreement with Abbott Laboratories for the development of Abbott's next-generation fenofibrate ABT-335 ; and Crestor in a single pill, fixed-dose combination treatment to target all three major blood lipids LDL-C `bad cholesterol', HDL-C `good cholesterol' and triglycerides. We may not complete these types of transactions or collaborative projects in a timely manner, on a cost-effective basis, or at all, and may not realise the expected benefits of any acquisition, licensing arrangement or strategic alliance. For example, in April 2007, we terminated our licensing and collaboration agreement with AtheroGenics, Inc. following the discontinuation of the development of AGI-1067 an investigational anti-atherosclerotic agent for the potential treatment of patients with coronary artery disease ; due to its failure to meet its target product profile. Other companies may also compete with us for these strategic opportunities. When we are able to complete these transactions, the success of these types of arrangements whether already existing or to be entered into in the future ; is largely dependent on the technology and other intellectual property acquired from a business or contributed from our strategic partners and the resources, efforts and skills of our partners. Disputes and difficulties in such relationships are common, often due to conflicting priorities or conflicts of interest. The benefits of these alliances would be reduced or eliminated should strategic partners terminate the agreements; fail to devote sufficient financial or other resources to the alliances; suffer negative outcomes in intellectual property disputes; fail to perform their obligations as expected; or impose controls and commercial limitations over the marketing and promotion of products developed under that collaboration. Also, under many of our strategic alliances, we make milestone payments well in advance of commercialisation of products, with no assurance that we will ever recoup those payments. If these types of transactions are unsuccessful, this may have a materially adverse effect on our financial condition and results of operations. References 1. Dutch SPC of Selektine. cbg-meb.nl version January 23, 2003 ; . 2. Dutch SPC of Zocor. cbg-meb.nl version December 15, 2000 ; . 3. Dutch SPC of Lipitor. cbg-meb.nl version April 17, 2002 ; . 4. Dutch SPC of Lipobay . cbg-meb.nl version July 12, 2000; access date May 21, 2001 ; . 5. Dutch SPC of Lescol . cbg-meb.nl version April 20, 2001 ; . 6. Dutch SPC of Crestor . cbg-meb.nl version May 7, 2003 ; . 7. De Gier JJ, Huyts JCPM: Commentaren medicatiebewaking Pharmacom Medicom. Houten: Stichting Health Base, 2002: IA 136. 8. Dutch SPC of Isoptin. cbg-meb.nl version March 20, 2000 ; . 9. Dutch SPC of Tildiem . cbg-meb.nl version July 22, 1999 ; . 10. Greenblatt DJ, von Moltke LL, Harmatz JS, Shader RI: Human cytochromes and some newer antidepressants: kinetics, metabolism, and drug interactions. J Clin Psychopharmacol 1999 Oct; 19 5 Suppl 1 ; : 23S-35S. 11. Chen XP, Tan ZR, Huang SL, Huang Z, Ou-Yang DS, Zhou HH: Isozyme-specific induction of low -dose aspirin on cytochrome P450 in healthy subjects. Clin Pharmacol Ther. 2003; 73: 264-71. Mousa O, Brater DC, Sundblad KJ, Hall SD: The interaction of diltiazem with simvastatin. Clin Pharmacol Ther 2000; 67: 267-274. Kantola T, Kivisto KT & Neuvonen PJ: Erythromycin and verapamil considerably increase serum simvastatin and simvastatin acid concentrations. Clin Pharmacol Ther 1998; 64: 177-182. Williams D, Feely J. Pharmacokinetic-pharmacodynamic drug interactions with Hmg -CoA reductase inhibitors. Clin Pharmacokinet 2002; 41 5 ; : 343-70. Astrazeneca collaboration - on july 5, 2006, abbott announced a collaboration with astrazeneca to jointly develop and market a single-pill, fixed-dose combination therapy of crestor with either tricor or abbott's proprietary next-generation fenofibrate abt-335 ; , currently in phase iii clinical development.

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